Monday, 2 August 2021

JULY i

JULY ASSESSMENT

Question 1

Case-1


Question 1: Competency tested for Peer to peer review and assessment : 

Please go through one student's entire answer paper frik kho kom this link, the one who is closest to your own roll number :




and share your peer review of each answer with your qualitative insights into what was good or bad about the answer. 


1.case : https://amishajaiswal03eloggm.blogspot.com/

question : 1) What is the evolution of the symptomatology in this patient in terms of an event timeline and where is the anatomical localization for the problem and what is the primary etiology of the patient's problem?

Review:I agree with the diagnosis because the patient works in paddy fields so he is affected with the allergens in the paddy field .The flow chart answered in this question is appropriate and gives the  complete symptomatology .

My review-

This is a case of a patient with cervical Myelopathy.patient came to hospital with complaints of weakness of all 4 limbs preceeding alcohol binge and fall.

In the answers

Condition of Myelopathy hand is defined clearly and pyramidal tracts are also mentioned.. It would be better if used a picture of this condition. Finger escape condition is explained and the nerves and muscles involved are also explained. And how finger escape is seen in cervical Myelopathy is also explained. Hoff mans reflex is also explained.. The explanation part is good.. But if diagrams are inserted it would be much more easier for understanding.


2 .case : https://avulanikhil09.blogspot.com/

Question: could chronic alcohlism have aggravated the foot ulcer formation ?if yes and why ?

Review : the answer for this explained is true and due chronic alcoholism ➡️ diminished immune response ➡️leads to ulcers 

My review-

informative and easy to comprehend to the point answers and very brief ,the usage of flow charts and diagrams is well used the usage of text can be better and the overall elog was well written.


3. case:  https://bejugamomnivasguptha.blogspot.com/

Question :  Is there any relationship between occurrence of seizure to brain stroke. (3 m)

Review : it is obviously true that shock may lead to seizures .

Shock ➡️scar formation in brain ➡️it disrupt electrical signal ➡️it leads to seizures .so the explanation in this answer is exactly true 

My review-

Patient came to opd with chief complaints of palpitations, pedal oedema, chest heaviness, radiating pain along the left upper limb. Patient was diagnosed with cervical spondylosis and recurrent hypokalemic paralysis.

About the answers given..anatomical localisation is mentioned and primary etiology is discussed deeply.. And causes of the symptoms are explained and possibility of other diseases are mentioned. Tables are used to explain clearly.. Risk factors of hypokalemia are mentioned and ecg changes in hypokalemia are explained with diagram. Usage of tables and diagrams made this presentation a bit more effective.



4.case :https://daddalavineeshachowdary.blogspot.com/

Diagnosis : it is a case of  55 yr old female with viral pneumonia  secondary to covid 19 and Dm 

Review : The case discussion is very good and the treatment is explained in the right order .

My review-

This is a case of acute coronary syndrome. The patient came to OPD with shortness of breath . She is known case of HTN and DM. The review is very well written in the form of nice flow chart about her past illness to present illness. And risk factors of DM and HTN point to a cardiac origin is also explained. In the review indications and contradictions of PCI is very well explained. The patient is treated for all her symptoms.

Ioi

5.Case :https://budigesaikiran14.blogspot.com/

Diagnosis : alcohol induced cerebellar ataxia 

Review : Reasons for cerebellar ataxia is very well discussed .

It is due to damage to GABA -A receptor impaired glucose metabolism vit B1 deficiency .the way of answering a question is so attractive and it is easy to understand .

My review-

The symptomatology has been explained in a chronological order of occurrence.

The primary etiology of the patient has been mentioned with the necessary finding along with the pictorial representation.

The anatomical location of the problem has been described.

The mechanism of action was very well explained with the action of the pharmacological drugs administered.

The answers for the given questions were commendable along with the usage of statistics


6.case: https://aniganikavya06.blogspot.com/

 Diagnosis :Acute exacerbation of COPD associated with right heart failure and bronchiectasis.

Review :  What is the evolution of the symptomatology in this patient in terms of an event timeline and where is the anatomical localization for the problem and what is the primary etiology of the patient's problem?

 Anatomical Location of problem-Lungs- Bronchi and bronchioles

  Primary etiology- Usage of chulha since 20years

Answer explained is appreciable .every thing is really so point to point and easily understandable 

My review-In this answer the etiology of the patient's problem is COPD due to conseqeunce of using chulha but I think that the problem is also with allergy while she is working in the paddy fields. The patient is suffering from the shortness of breathing from long time due to the inhalation of smoke and that caused damage to the lungs,thus the patient is susceptable to the lung infections.

 In this link, the evaluation of symptomatology was explained well. Pharmacological and Non pharmacological interventions were mentioned but not in a detailed manner. The placebo effect was also not explained properly. I think the overall case and the answers to the questions could have been explained properly.


Case : 7 https://02shishirareddy.blogspot.com/

 Diagnosis : Alcohol induced dementia 

Review : Causes for electrolyte imbalance: 

                       Respiratory acidosis with metabolic alkalosis(due to renal compensation) in AECOPD patientswith chronic hypercapnia is the usual cause ofhypochloremia in these patient Commoncauses ofderanged serum sodium levels include hyperglycemia, use of thiazides or nonsteroidal anti-inflammatory drugs, congestive cardiac failure, chronic renal failure, and low dietary salt intake.

 Common causes of hypokalemia include diarrhea, laxative abuse, vomiting, certain diuretics, drugs like insulin, β2agonists, and theophylline.20 Thus, COPD patients per

se are predisposed to electrolyte imbalance. In turn electrolyte imbalance can cause respiratory muscle weakness, cardiac arrhythmia, low cardiac output. 

                    Thus the presence of electrolyte imbalance leads to significantly poor outcome among COPD patients.It is very interesting to know about the acidosis and very happy to read and get the knowledge about respiratory acidosis .it is really very helpful .

My review-

The history of present and past illness is coherent and well established. The videos are great for understanding the case. The case sheet has captured all the relevant data in the right order. Correct terminology was used. The reports of all the lab investigations conducted were given. The pharmacological intervention was elaborate and explained well. 

The symptomatology with anatomical localization and primary etiology was explained very well. The explanation was done with help of diagrams which made it easier to understand. The treatment part was also explained with all the drugs and injections that were given to the patient. The causes that are explained for acute exacerbation and for electrolyte imbalance were appropriate. At last all the questions were answered and case was handled well.


Case :8 https//savanthreddy.blogspot.com/

Diagnosis : INFECTIOUS DISEASE (Mucormycosis) 

Review : What are the postulated reasons for a sudden apparent rise in the incidence of mucormycosis in India at this point of time?

I think it is because of the use steroids in an uncontrolled manner.

COVID 19 is not the first disease where steroids are used as one of the main stay of treatment (in severe cases) but what has gone wrong in the recent time is even the lay man who got infected with Corona started using steroids right from the day 1 with no monitoring by a specialised physician.And they are not following any specific course of treatment.As many of them are also diabetic patients the are getting succumbed to mucormycosis.And it is not ending with mucormycosis but various other fungii are emerging to cause an epidemic.

And another reason for rise in mucormycosis cases may be the usage of tap water in oxygen concentrators.As most of the patients are being diagnosed of Covid in late stages in the second wave many of them Are requiring oxygen concentrators for supplemental oxygenation.And due to Lack of the people are using tap water instead of distilled water which serves as reservoir for the growth of mucormycosis.So this may be one of the reason for rise in mucormycosis cases. I feel really happy by reading this new information about the disease that is happening in our india and it is very well explained with the good image and reason behind the this epidemic 

My review-

Evolution of symptomatology was listed in a chronological order which was easy to understand. Only non-pharmacological interventions were explained, could have been better if pharmacological interventions were also included. The cause for acute exacerbation in this case was not explained in detail. Overall, the answers to the questions could have been answered in a  better way by adding some of the elements as mentioned above and explaining them in detail.



Case 9 : https://gsuhithagnaneswar.blogspot.com/?m=1

Diagnosis : cervical spondylosis Recurrent hypokalemic paralysis 

   Review : What are the reasons for recurrence of hypokalemia in her? Important risk factors for her hypokalemia? ➡️Since the patient complains of oedema the drugs used to relieve it such as diuretics can cause hypokalemia 

The patient also no albumin which is a cause for both oedema and hypokalemia 

The risk factors including 

1.    Alcohol use(excessive

2.    Chronic kidney disease

3.    Diabetic ketoacidosis

4.    Diuretics (water retention reliever

5.    Excessive laxative 

6.    Folic acid deficiency

7.    Primary aldosteronism  

8.    Some antibiotic uses 

It is point to point and easy everything is easily reviseble .happy to read about this new case 

My review-

The evolution of symptoms was explained in tabular form chronologically,it is quite good.Pharmacological and non-pharmacological interventions of each drug are explained with their mechanisms.But there are no pictures and flow charts.The journal link was also updated for more info.


Case 10 :   https://blendedasessmentmadhukumar.blogspot.com/

Diagnosis : inferior wall MI with uncontrolled sugars with k/c/o DM since 8yrs.

Review : What is the evolution of the symptomatology in this patient in terms of an event timeline and where is the anatomical localization for the problem and what is the primary etiology of the patient's problem?

➡️Chest pain- since 3days

Giddiness - since morning

Profuse sweating- since morning

Anatomical location of the lesion is inferior wall of the heart.

Etiology: 

Smoking

Diabetes mellitus

Hypertension

My review-

The symptomatology in the patient in terms of an event, based on timeline was explained properly. The pharmacological and Non-pharmacological interventions used by this patient were appropriate and in detail. All the questions were also answered but could have been listed the causes properly.


Q2-4

Patient centered data around the theme of renal failure patients with AKI, CKD and acute on CKD, 

captured by students from 2016 and 2019 batch in the links below

Patients with low back ache and renal failure

Q2) Share the link to your own case report of a patient that you connected with and engaged while capturing his her sequential life events before and after the illness and clinical and investigational images along with your discussion of that case. 

http://arunrollno46.blogspot.com/2021/07/aki.html

I have helped my juniors in taking histories and guided them in preparing elogs and presenting them in groups and in 2-4 sessions.


Q3) (Testing peer review competency of the examinees) :

Please go through the cases in the links shared above and provide your critical appraisal of the captured data in terms of completeness, correctness and ability to provide useful leads to analyze the diagnostic and therapeutic uncertainties around the cases shared.

AKI 

https://laharikantoju.blogspot.com/2021/07/58-year-old-male-patient-elog-lahari.html?m=1

Over view 

A 58 year old male patient came to casualty with chief complaints of:

- lower abdominal pain: 1 week

 -burning micturation:1week

- low back ache after lifting weights

-dribbling / decrease of urine out put:1week

-fever :1 week

- SOB , rest :1week  

  Apprisal

Case history was taken well and examination was very well done... Sequential evaluation of case is apprisiable 

Negative points 

It would be better if fever chart is added as it was treated with strict temp and IO monitoring as it would be better understood improvement of the case was not well mentioned

My Analysis

 This is a case of Acute kidney injury( AKI) 2° to UTI, associated with Denovo - DM -2

With ? Right HEART FAILURE,

With K/C/O - HTN ( Not on Rx)

-AKI causes a build-up of waste products in your blood and makes it hard for your kidneys to keep the right balance of fluid in your bodyand return of creatinine to the base line and symptoms less then 3 months indicating it to be a AKI

Acute on CKD :

http://srinaini25.blogspot.com/2021/07/srinaini-roll-no-33-3rd-semester-this.html

Over view 

A 75yr old male patient ,labourer by occupation ,came to casuality with Cheif complaints of 

• Lower backache since 10days

• dribbling of urine since 10days

• Pedal edema since 3days 

• SOB at rest since 3days 

• Increased involuntary movements of both upper limbs since 10days .

Apprisal

I would not find any points to be highlighted . History was taken well 

Negative points

There are no clinical pics of the symptoms like pedal edema.

Proper chronological order of symptoms apperance was not done 

Fever chart was not included.

No IO charting was done though it was told it should be strictly monitored

My Analysis

This is case of 

Acute renal failure (intrinsic)

 Grade 1 L4-L5 Spondylodiscitis, Multifocal infectious Spondylodiscitis

Hyperuricemia 2° to Renal failure 

Uraemia induced tremors( resolved)

Delerium 2° to septic /Uremic encephalopathy (resolving)

CKD :

https://krupalatha54.blogspot.com/2021/07/a-49-yr-old-female-with-generalized.html?m=1

Over view

A 49 yr old female , mother of 2 children, who is a house wife, apparently asymptomatic 13 yrs ago and then she noticed mass per anum with bleeding , went to hospital and diagnosed as haemorrhoids and got operated.

- Since 3 yrs she has history of muscle aches, for which she is using NSAIDs.

- She has h/o fever 20 days back, got treated in the local hospital, and 

- Since 20 days she has generalized weakness.

- She also has h/o vomitings since 3 days, with food as content, non - projectile , non bilious.

Apprisal

History was taken well.

Good lab work clear evaluation was done 

Negative points

There are no clinical pics of the symptoms like pedal edema.

Proper chronological order of symptoms apperance was not done 

Fever chart was not included.

No IO charting was done though it was told it should be strictly monitoredit would have been better if urine was sent for eosinophils for interatial disease

My Analysis

This is  case of CKD ?

 Chronic interstitial nephritis secondary to plasma cell dyscariasis, (multiple myeloma - 70% plasmacytosis).

Patient with coma and renal failure 

https://ananyapulikandala106.blogspot.com/2021/06/a-35yr-old-female-elog.html

Overview

A 35 yr old female with Fever and Diarrhea since 5 days( 4 to 5 times a day with blood discharge).

Back pain( 5 days ago) with abdominal pain and chest pain.

Apprisal

Very well presented 

With good fever charting with all the necessary information.

History was taken detailed way 

Follow up was good 

All the tests were properly done 

Negative

I could not find the negative data in the elog 

My analysis

It could be the hypoxia which could have caused the permanent brain damage which was the reason for her vegetative state . Subjectively she was told better but objectively no improvement was Seen. Hospitalisation has increased the infection to the bed sore it would have been better if discharged early as it was permanent damage and was impossible to treat anyway.

Q4: Testing scholarship competency of the examinees ( ability to read comprehend, analyze, reflect upon and discuss captured patient centered data as in their 'original' answers to the assignment for May 2021):


Please analyze the above linked patient data by first preparing a problem list for each patient (based on the shared data) and then discuss the diagnostic and therapeutic uncertainty around solving those problems. Also include the review of literature around sensitivity and specificity of the diagnostic interventions mentioned and same around efficacy of the therapeutic interventions mentioned for each patient. 

Analysis the data

 https://ananyapulikandala106.blogspot.com/2021/06/a-35yr-old-female-elog.html 

Analysis of  A 35 yr old female with Fever and Diarrhea since 5 days( 4 to 5 times a day with blood discharge).

Back pain( 5 days ago) with abdominal pain and chest pain

Vegetative state of the patient could be due to  hypoxia which could have caused the permanent brain damage which was the reason for her vegetative state . Subjectively she was told better but objectively no improvement was Seen.

https://pubmed.ncbi.nlm.nih.gov/19362767/

Link supporting the data 



Intermittent Fever spikes can be explained due to the bed sores clearly explained through culture of the sores 




Sepsis might be the reason for encephalopathy by altering the blood brain barrier 


https://www.hindawi.com/journals/amed/2014/762320/


Q 5) Testing scholarship competency in  

logging reflective observations on your concrete experiences of this last month : (10 marks) 


Reflective logging of one's own experiences is a vital tool toward competency development in medical education and research. 

The telemedical learning from the hospital has been a new experience and we  learnt quite lot of things through reflective observation during lockdown.  it's a bit challenging as we have just entered internship ,We have learnt elogging of the cases in a very short span of time and made juniors do so. I could answer the questions from juniors easily as I have been part of many discussion in ICU and wards . By doing this assignment I could view many cases and many case scenarios through which I learned many things


JUNE ASSESSMENT

JUNE ASSESSMENT

 Question 1: Competency tested for Peer to peer review and assessment : 


Please go through one particular answer of ten students in this link:
and share your peer review of each answer with your quantitative marking input as well as qualitative insights into what was good or bad about the answer. 

 1. PULMONOLOGY

https://soumyanadella128eloggm.blogspot.com/2021/05/a-55-year-old-female-with-shortness-of.html

1) Evolution of symptamatology


20yrs ago- 1st attack of shortness of breath

From then, every year in January, episode lasts for a week

12yrs ago- admitted to hospital with severe shortness of breath


Current episode:

Started 30 days back, dyspnoea on exertion, relieved on rest, 

But since 2 days, dyspnoea even at rest


Possible explanation: attacks of dyspnoea can be triggered by winter season, vegetative matter, dust, pollen ( provided dyspnoea occurred at the time of working in rice fields) 

Possible etiology: vegetative dust (RICE) 

On investigation: upon CT, lower airway problem is seen

Other associated symptoms :

   1. Pedal edema since 15 days up till ankle level

   2. Facial puffiness since 15 days


MY OPINION AND ANALYSIS:-chief complaints:-

Being SOB,facial puffiness and pedal edema

TO RULE OUT CARDIAC CAUSE OR RENAL CAUSE

As investigations and clinical examination show

BRONCHIECTASIS

AND ACUTE EXACERBATION OF COPD..

Im convinced with her analysis and review



Question 2-4: 

Patient centered data 
captured by students from 2016 batch in the link below:

MULTISYSTEM:


CNS :


Renal :


Captured by one student from 2017 batch in the link below :

Captured by one student from 2019 batch in the link below :

Abdominal : 




MY ANALYSIS OF THE FOLLOWING CASES:-



1.https://casescape.blogspot.com/2021/06/acute-kidney-injury-secondary-to.html?m=1

Overview:-
pt is diagnosed with AKI SECONDARY TO UROSPESIS
IN 2019
Came with similar complaints in june 2021
Could probably be a case of AKI ON CKD
RATHER THAN AKI 


APPRAISAL:-GOOD CHARTING OF TRENDS OF SERUM CREATININE AND TLC COUNTS


NEGATIVE POINTS:-
COULD HAVE SHARED DEINDENTIFIED IMAGES OF THE PATIENT SO AS TO GIVE A CLEAR AND DETAILED OPINION ABOUT EXAMINATION 
COULD HAVE SHARED A MORE DETAILED HISTORY OF HER HISTORY AFTER DIALYSIS SESSIONS FROM 2019 to 2021 
FEVER CHART SHOULD HAVE BEEN SHARED



ANALYSIS:-THIS COULD BE AKI ON CKD
RATHER THAN AKI ALONE

2. CNS :


OVERVIEW:-

chief complaints of sudden fall followed by weakness of both the lower limbs (paraplegia) and loss of hand grip 10 days back, associated with bowel and bladder incontinence.

He is a known case of TB since 1month and on ATT - HRZE

Probably could be pott’s spine secondary to TB

APPRAISAL:-
Well
Presented case with detailed info about patient and imaging reports

NEGATIVES:-
Fever chart should have been shared and SOAP format of patients everyday progress


ANALYSIS:-Quadreparesis secondary to infectious spondylitis of C4, C5, C6, C7 and D1 with Epidural abscess at C5 - C6 level.
Clinically CNS:
Speech - normal
No signs of meningeal irritation
                  Right.        Left
Tone. UL.  N.              N
           LL  increased. Increased
Power UL.  4/5.         4/5
             LL.  1/5.          1/5
Cranial.nerves.  : Intact
Sensory system : normal
Reflexes:
            Right.         Left
Biceps. 3+.             3+
Triceps. 3+.            3+
Supinator. 2+.         2+
Knee.          3+.         3+
Ankle.        3+.          3+

Plantar: extensor
I dont these findings actually are supportive of the daignosis
Because in a case of quadriparesis
The examination findings donot correspond

3. Renal :

OVERVIEW:-Complaint of Altered Sensorium (Hypo active):It's since  Morning,lethargy.
History of fever 10 days back,lasted for 3 days,(Outside Creatinine ?11-14.8)
Followed by Pedal edema with Anasarca with Shortness of breath present even at rest .
(Outside Creatinine reports 11)

He is a known case HYPERTENSION:5 YEARS and on T.STAMLOBETA OD.
He is also a known case of CHRONIC KIDNEY DISEASE:5 YEARS and on Conservative Treatment
(Outside Creatinine =? 3.2 mg/dl,5years back).

Present creatinine of 20mg/dl and previous baseline creatinine above normal 
It is a case of AKI ON CKD with Level of consciousness:Drowsy but Arousable (Hypoactive, delayed response to commands).
Signs of meningeal irritation:No neck stiffness,no kerning's sign.
Higher mental functions: Intact
Reflexes: Present.
Suggestive of UREMIC ENCEPHALOPATHY (since urea levels being 340mg/dl)


NEGATIVES:-could have shared more clinical
Images (deidentified)
And fever chart should have been shared
Recent trends of RFT could have been shared



APPRAISAL:-Good presentation of the case with proper history and treatment plan



ANALYSIS:-
Since baseline creatinine levels are above normal
And current compliants of anasarca
With raised creatinine of 20mg/dl and urea of 340mg/dl
With a drowsy state suggests AKI ON CKD WITH UREMIC ENCEPHALOPATHY

4. CVS :


OVERVIEW:-A 70 year old with complaints of Distension of abdomen and shortness of breath Grade-3 since 5days with ECG  Showing Atrial fibrillation

And 2D Echo ;

Akinetic segment in LAD territory with EF 35%  and RVSP 100mmhg

B/L pleural effusion,mild pericardial effusion.

Diagnosed as

HFrEF with Atrial fibrillation 2 to ?IHD

Her biochemical report showing severe hyperthyroidism possibly relating to her refractory Atrial fibrillation 

APPRAISAL:-well
Presented case with history and detailed findings in ECG and 2D ECHO

NEGATIVES:-Could have shared the BP CHARTING AND FEVER CHARTING with deidentified clinical
Images of patient.

ANALYSIS:-A 70 year old with complaints of Distension of abdomen and shortness of breath Grade-3 since 5days with ECG  Showing Atrial fibrillation investigations showing

HFrEF with Atrial fibrillation 2 to ?IHD

And Her biochemical report showing severe hyperthyroidism possibly relating to her refractory Atrial fibrillation 



Q2) Share the link to your own case report of a patient that you connected with and engaged while capturing his /her sequential life events before and after the illness and clinical and investigational images along with your discussion of that case. 

http://arunrollno46.blogspot.com/2021/07/aki.html


Q 5) Testing scholarship competency in  
logging reflective observations on your concrete experiences of this last month : (10 marks) 

Reflective logging  of one's own experiences is a vital tool toward competency development in medical education and research. 

THIS POSTING HAS BEEN A LEARNING EXPERIENCE ON ANALYSING THE LOGGING THE PATIENT DATA AND GETTING TO DISCUSS THE CASE WITH MY COLLEAGUES AND PGS AND THE FACULTY AND I HAVE LEARNED ABOUT THE VARIOUS TREATMENT PROTOCOLS AND HANDS ON EXPOSURE REGARDING SAMPLE COLLECTION, ABG, RYLES TUBE AND FOLEYS CATHETER INSERTION, ASCITIC TAP, PERFORMING DIALYSIS SESSIONS , PERFORMING CPR, OBSERVING INTUBATION, CENTRAL VENOUS CATHETERIZATION AND PRESENTING THE CASES IN 2-4 sessions.

Sunday, 1 August 2021

heart failure

A 60-year-old male patient resident of bhongiri came to the OPD with chief complaints of shortness of breath (grade3-4 according to NYHA classification )since yesterday ;complains of abdominal distension since yesterday ,decreased urine output since yesterday , B/l  Pedal edema since   Yesterday and  burning micturition since two days .

History of presenting illness: –
Patient was apparently a symptomatic till yesterday then he developed shortness of breath (grade3-grade4) according to NYHA with Orthopnoea not associated with PND , Palpitations chest pain.
Complains of abdominal distension not associated with nausea or vomiting.
Complains of bilateral Pedal edema of pitting type up to the knee.
Complains of decreased urine output since yesterday complains of burning micturition.


Known case of alcohol Consumption – occasionally(once a week- 90ml whiskey)
k/c/o htn on irregular medication
Not a known case of diabetes mellitus 
past history:- 
he was admitted in july 2020 with similar complaints and was diagnosed with acute decompensated heart failure with moderate MR.
He stopped his medications since 3 months.
Personal history: –
Marital status-married
Appetite – normal 
Diet-mixed
Bubble and bladder – regular
Addictions – alcohol consumer occasionally, stopped since one year.
No Known allergies.
Family history: –
Insignificant
General physical examination –
Patient is conscious coherent and cooperative and examine well lit room.
Pedal edema : ++
No signs of pallor, Cyanosis, clubbing, Icterus, lymphadenopathy .
Temperature : 98.5° F
Pulse rate: 94 BPM
Respiratory rate: 18 cycles per minute
BP: 120/ 70 mm of Hg
Spo2: 96%
Grbs : 115mg%
CVS:-S1, S2 heard ; Pan systolic murmur heard in mitral area.
apex beat: left 5th ics
elevated jvp
orthopnea:+
no thrill
pulse: rate:94bpm
            rhythm: regularly irregular

Respiratory system: –
Dyspnea: present (grade3-4)
Position of trachea – Central
breath sounds – BAE plus
Crepitations heard in lt. MSA, IAA
Per Abdomen: –
Shape of the abdomen – obese
Tenderness – absent
No palpable mass , no organomegaly
Hernia notifies intact, no free fluid ,no bruits.
bowel sounds : present

Central nervous system examination –:
Level of consciousness – alert
Speech-normal
No signs of Meningeal 
Reflexes normal.
 
ecg findings:. inverted p waves in lead 1 ,2 ,3 and ventricular premature complexes seen in V4, V5, V6.
junctional rhythm with occasional vpc.

Provisional diagnosis:- acute decompensated heart failure